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Peer reviewedOpen accessLeishmaniasis

Bi-directional highways and super-seeder tissues underpin parasite dissemination in experimental Leishmania donovani infection

Nature Communications·

Ciara Loughrey, Juliana B. T. Carnielli, Shoumit Dey, Helen Ashwin, Sally James, Lesley Gilbert, Samantha Donninger, Alastair Droop, Grant Calder, Karen Hogg, Jon Pitchford, Jeremy C. Mottram, Paul M. Kaye

DOI
10.1038/s41467-026-77072-4
PMID
PMCID
OpenAlex
W7204261393
Study type
Ecological study
Publisher
Springer Science and Business Media LLC
Article type
journal-article
Integrity
current

Why this research matters now

Clarifying these internal migration patterns may help explain recurring infections and therapeutic resistance, offering conceptual guidance for managing disease propagation.

01

Structured evidence summary

Research question

The investigation seeks to clarify how Leishmania donovani migrates through various anatomical compartments and selects specific tissues during infection.

Study design

Researchers conducted an experimental trial using a laboratory mouse model, integrating a genetically tagged parasite collection with computational ecological methods to track infection spread.

Population and setting

The analysis focused on a controlled murine cohort subjected to experimental parasitic infection.

Main findings

Analysis revealed dense network connections between internal organs and cutaneous layers, indicating constant reciprocal movement. Specific anatomical regions were identified as major amplification centers that drive widespread infection. Furthermore, alterations in host immunity triggered shifts in the primary pathways used by the parasite to travel.

Public-health relevance

Clarifying these internal migration patterns may help explain recurring infections and therapeutic resistance, offering conceptual guidance for managing disease propagation.

Important limitations

The experimental approach depends entirely on rodent subjects, which inherently limits direct translation to human clinical presentations and immune responses.

GIDS interpretation

This work establishes a mechanistic baseline for tracking pathogen distribution within hosts, providing contextual reference points for future epidemiological modeling. It highlights theoretical transmission corridors rather than reporting active field detection events.

02

Related GIDS surveillance

Literature context does not validate, explain, or change a surveillance signal. Exact and contextual relationships are shown separately.

03

Evidence relationships

This article has 7 auditable classifier relationships to diseases, places, topics, and study design.

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