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Peer reviewedOpen accessH5N1Influenza

Experimental adaptation of an influenza H5 HA confers respiratory droplet transmission to a reassortant H5 HA/H1N1 virus in ferrets

Nature·

Masaki Imai, Tokiko Watanabe, Masato Hatta, Subash C. Das, Makoto Ozawa, Kyoko Shinya, Gongxun Zhong, Anthony Hanson, Hiroaki Katsura, Shinji Watanabe, Chengjun Li, Eiryo Kawakami, Shinya Yamada, Maki Kiso, Yasuo Suzuki, Eileen A. Maher, Gabriele Neumann, Yoshihiro Kawaoka

DOI
10.1038/nature10831
PMID
22722205
PMCID
PMC3388103
OpenAlex
W2112170460
Study type
Journal article
Publisher
Springer Science and Business Media LLC
Article type
journal-article
Integrity
current

Why this research matters now

Characterizing the genetic requirements for avian influenza hemagglutinin to support mammalian spread aids in forecasting pandemic risks and directing resource allocation for medical countermeasures.

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Structured evidence summary

Research question

What molecular alterations in the hemagglutinin protein enable a reassortant influenza virus to achieve respiratory droplet spread in mammals?

Study design

Controlled laboratory experiment employing a ferret model to evaluate the transmission capacity and disease severity of a genetically modified reassortant influenza strain.

Population and setting

Ferrets utilized as a mammalian surrogate system to assess viral replication, receptor affinity, and airborne dissemination.

Main findings

A reassortant virus containing an adapted H5 hemagglutinin segment with four specific mutations achieved efficient droplet transmission between ferrets. The pathogen demonstrated robust replication and human-type receptor preference while producing mild pulmonary damage without lethality. Authors caution that additional genetic components beyond these four mutations likely influence complete mammalian transmissibility.

Public-health relevance

Characterizing the genetic requirements for avian influenza hemagglutinin to support mammalian spread aids in forecasting pandemic risks and directing resource allocation for medical countermeasures.

Important limitations

The authors explicitly note uncertainty regarding whether the identified four mutations alone would confer transmissibility to a fully avian H5N1 strain, and acknowledge that other viral gene segments likely influence mammalian spread. Because the evaluation relies solely on the supplied abstract and metadata, the original manuscript must be consulted for comprehensive methodological detail and decision-grade interpretation.

GIDS interpretation

This work supplies mechanistic insights into hemagglutinin-driven cross-species transmission, establishing a reference point for investigators monitoring influenza genetic drift and evaluating emerging variant characteristics.

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Related GIDS surveillance

Literature context does not validate, explain, or change a surveillance signal. Exact and contextual relationships are shown separately.

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Evidence relationships

This article has 9 auditable classifier relationships to diseases, places, topics, and study design.

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