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Peer reviewedOpen accessTuberculosis

The bacterial pH gradient contributes to persistence in Mycobacterium tuberculosis

mBio·

Hassan E. Eldesouky, Kristin N. Adams, Justin K. Brache, Laarni Kendra T. Aguila, Mariana Garcia, Enming Xing, Pui-Kai Li, David R. Sherman

DOI
10.1128/mbio.01692-26
PMID
PMCID
OpenAlex
W7203700488
Study type
Journal article
Publisher
American Society for Microbiology
Article type
journal-article
Integrity
current

Why this research matters now

Findings suggest that pharmacological disruption of the bacterial pH gradient is a rapid chemical tool to study persister biology in TB, with potential broader relevance to other infectious diseases.

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Structured evidence summary

Research question

The study investigates how chemical perturbation of the bacterial pH gradient and related proton motive force components influences drug-tolerant persistence in Mycobacterium tuberculosis.

Study design

A laboratory-based screening study using 2,336 FDA-approved drugs to identify compounds that modulate Mtb persistence, followed by targeted microbiological assays, transcriptomic profiling, and targeted gene knockdowns.

Population and setting

In vitro experimental work using Mycobacterium tuberculosis cultures; no human participants, clinical settings, or geographic field populations are described.

Main findings

Niclosamide protected Mtb against bactericidal doses of isoniazid, rifampicin, and other standard TB drugs. Disruption of the pH gradient with intracellular acidification, rather than disruption of membrane potential, was required to induce tolerance, and the protective effect was tunable by external pH. Transcriptomic analysis and knockdowns linked three Mtb-specific genes to either promoting or mitigating the tolerant state.

Public-health relevance

Findings suggest that pharmacological disruption of the bacterial pH gradient is a rapid chemical tool to study persister biology in TB, with potential broader relevance to other infectious diseases.

Important limitations

This summary is limited to the supplied single-article abstract and metadata; the original paper is required for decision-grade interpretation. The abstract does not explicitly state limitations of the study.

GIDS interpretation

The article is discoverable through TB- and treatment-related classifiers within GIDS; it provides context on laboratory models of Mtb drug tolerance and does not itself constitute a live surveillance signal.

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Related GIDS surveillance

Literature context does not validate, explain, or change a surveillance signal. Exact and contextual relationships are shown separately.

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Evidence relationships

This article has 6 auditable classifier relationships to diseases, places, topics, and study design.

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