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Peer reviewedOpen accessMalaria

Multi-platform evidence of GSDMD-dependent pyroptosis in malaria with a potential role for TLR7

Frontiers in Immunology·

Yumei Gong, Yiwei Liu, Longkun Li, Jialin Geng, Ying Huang, Lixin Luo, Xinyue Wang, Long Xu, Yongtao Zhang, Xingfei Pan, Lu Li, Shan Zhao, Jun Huang

DOI
10.3389/fimmu.2026.1862560
PMID
PMCID
OpenAlex
Study type
Journal article
Publisher
Frontiers Media SA
Article type
journal-article
Integrity
current

Why this research matters now

The study addresses macrophage-associated inflammation in malaria and identifies TLR7-mediated regulation of pyroptosis as a potential area for further investigation.

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Structured evidence summary

Research question

The study investigated pyroptosis pathways in splenic macrophages during Plasmodium infection and whether a TLR7 agonist could regulate that process.

Study design

An integrated study combining clinical transcriptomic analysis, a Plasmodium yoelii-infected mouse model, and in vitro macrophage assays.

Population and setting

The clinical component analyzed transcriptomic data associated with malaria patients; experimental components used P. yoelii-infected mice and macrophage cultures exposed to infected red blood cell lysates.

Main findings

Core pyroptosis genes GSDMD and CASP1/4/5 were significantly upregulated in clinical samples and correlated with myeloid expansion. P. yoelii infection was associated with canonical and non-canonical GSDMD-related pyroptosis in splenic macrophages, while infected red blood cell lysates induced Caspase-1/11 and GSDMD cleavage, membrane perforation, and inflammatory efflux in vitro. R848 administration was associated with reduced pyroptotic signaling, with a particularly strong effect on the Caspase-11/GSDMD non-canonical pathway.

Public-health relevance

The study addresses macrophage-associated inflammation in malaria and identifies TLR7-mediated regulation of pyroptosis as a potential area for further investigation.

Important limitations

The supplied single-article abstract does not provide explicit limitations. This summary is limited to the supplied abstract and bibliographic metadata and requires the original paper for decision-grade interpretation.

GIDS interpretation

The article is discoverable in GIDS through the topics of malaria and treatment. This classification provides topical context only and does not connect the study to a live surveillance signal.

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Related GIDS surveillance

Literature context does not validate, explain, or change a surveillance signal. Exact and contextual relationships are shown separately.

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Evidence relationships

This article has 5 auditable classifier relationships to diseases, places, topics, and study design.

about diseaseaddresses topicevaluates interventionstudied population settinguses study design