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Peer reviewedOpen accessInvasive pneumococcal disease

Pneumococcal lineages associated with serotype replacement and antibiotic resistance in childhood invasive pneumococcal disease in the post-PCV13 era: an international whole-genome sequencing study

The Lancet Infectious Diseases·

Stephanie W Lo, Rebecca A Gladstone, Andries J van Tonder, John A Lees, Mignon du Plessis, Rachel Benisty, Noga Givon-Lavi, Paulina A Hawkins, Jennifer E Cornick, Brenda Kwambana-Adams, Pierra Y Law, Pak Leung Ho, Martin Antonio, Dean B Everett, Ron Dagan, Anne von Gottberg, Keith P Klugman, Lesley McGee, Robert F Breiman, Stephen D Bentley, Abdullah W Brooks, Alejandra Corso, Alexander Davydov, Alison Maguire, Andrew Pollard, Anmol Kiran, Anna Skoczynska, Benild Moiane, Bernard Beall, Betuel Sigauque, David Aanensen, Deborah Lehmann, Diego Faccone, Ebenezer Foster-Nyarko, Ebrima Bojang, Ekaterina Egorova, Elena Voropaeva, Eric Sampane-Donkor, Ewa Sadowy, Godfrey Bigogo, Helio Mucavele, Houria Belabbès, Idrissa Diawara, Jennifer Moïsi, Jennifer Verani, Jeremy Keenan, Jyothish N Nair Thulasee Bhai, Kedibone M Ndlangisa, Khalid Zerouali, K L Ravikumar, Leonid Titov, Linda De Gouveia, Maaike Alaerts, Margaret Ip, Maria Cristina de Cunto Brandileone, Md Hasanuzzaman, Metka Paragi, Michele Nurse-Lucas, Mushal Ali, Naima Elmdaghri, Nicholas Croucher, Nicole Wolter, Nurit Porat, Özgen Köseoglu Eser, Patrick E Akpaka, Paul Turner, Paula Gagetti, Peggy-Estelle Tientcheu, Philip E Carter, Rafal Mostowy, Rama Kandasamy, Rebecca Ford, Rebecca Henderson, Roly Malaker, Sadia Shakoor, Samanta Cristine Grassi Almeida, Samir K Saha, Sanjay Doiphode, Shabir A Madhi, Shamala Devi Sekaran, Somporn Srifuengfung, Stephen Obaro, Stuart C Clarke, Susan A Nzenze, Tamara Kastrin, Theresa J Ochoa, Veeraraghavan Balaji, Waleria Hryniewicz, Yulia Urban

DOI
10.1016/s1473-3099(19)30297-x
PMID
31196809
PMCID
PMC7641901
OpenAlex
W2950614621
Study type
Genomic study
Publisher
Elsevier BV
Article type
journal-article
Integrity
current

Why this research matters now

Findings suggest emerging non-vaccine serotypes may be shaped by local antibiotic-selective pressures, highlighting the need for continued genomic surveillance to inform future vaccine design.

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Structured evidence summary

Research question

The study investigated pneumococcal lineages associated with predominant serotypes, mechanisms of serotype replacement following pneumococcal conjugate vaccine introduction, and antibiotic resistance characteristics of major lineages in the post-vaccine era.

Study design

An international whole-genome sequencing study analyzing 3,233 invasive pneumococcal disease isolates from six countries, with comparison between pre-vaccine and post-vaccine periods to assess serotype replacement and lineage contributions.

Population and setting

Children younger than 3 years from Hong Kong, Israel, Malawi, South Africa, The Gambia, and the USA, with isolates from laboratory-based surveillance programs spanning before and after PCV introductions.

Main findings

The five most prevalent serotypes in the PCV13 period varied by country. Serotype replacement was driven primarily by expansion of non-vaccine serotypes within vaccine-type lineages, with GPSC3 identified as a globally spreading lineage. Significant increases in penicillin and erythromycin resistance were observed among non-vaccine serotype isolates in the post-vaccine period.

Public-health relevance

Findings suggest emerging non-vaccine serotypes may be shaped by local antibiotic-selective pressures, highlighting the need for continued genomic surveillance to inform future vaccine design.

Important limitations

No explicit limitations were stated in the supplied abstract. Conclusions are therefore limited to the information available in the abstract and require review of the full original paper for decision-grade interpretation.

GIDS interpretation

This genomic surveillance study of invasive pneumococcal disease isolates across multiple countries provides contextual data on serotype distribution and resistance patterns in the post-PCV era. The findings represent population-level surveillance evidence and do not constitute a signal requiring immediate action.

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Related GIDS surveillance

Literature context does not validate, explain, or change a surveillance signal. Exact and contextual relationships are shown separately.

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Evidence relationships

This article has 12 auditable classifier relationships to diseases, places, topics, and study design.

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